Auto-immune diseases

Auto-Immune (AI) diseases are often quite disabling for the person affected. The cause is usually unknown but there may be a connection with mould.

Most of the time doctors are at a loss to explain the cause of AI disease and do not have the tools to investigate the role of mould in the disease process.

In a nutshell AI disease is about failures of the CD4+ class of lymphocytes (also known as the T-Helper cells type 1 and 2). The TH1 and TH2 cells live for many years directing and controlling the rest of the immune system. They could be called the "parent" cells and one of their crucial jobs is to control younger more powerful cells called TH17 cells. TH17 cells are so named because they produce a lot of Interleukin-17, a key immune signalling protein.

If TH1 and TH2 are the parents, the TH17 could be called the "hairy teenagers" of the immune system. Young and powerful they will pile into a fight against microbes with all guns blazing. However if they cannot be controlled by their parents they can cause serious tissue damage over time with the result that joints are destroyed in RA (Rheumatoid Arthritis), and crippling lesions occur in the central nervous system of MS sufferers.

The role of toxic mould

The author has found Aspergillusmould causing auto-immune disease involving the eye, bladder and joint lining. This mould produces numerous mycotoxins and volatile organic compounds (VOCs) that seem to affect the CD4+ and TH17 cells differently. The younger TH17 cells seem more resistant to these toxins and continue to operate while the older CD4+ cells become disabled and lose the ability to control the TH17 cells. This initially produces pain and inflammation but over time tissue destruction occurs leading to chronic disease changes. Those of rheumatoid arthritis are well known.

The older CD4+ cells are often operating at lower energy due to corruption of three important mechanisms:
1. The activity of the glycogen synthase enzyme can be greatly reduced by mercury and cadmium toxicity. Glycogen is the storage molecule for glucose held in the cell to feed into the mitochondria to produce ATP;
2. The production of ATP in the mitochondria can be affected by fungal mycotoxins, chemicals and heavy metals;
3. The DNA rings of the mitochondria can also become contaminated leading to a failure of mitochondrial reproduction within the cells. Ageing mitochondria are not replaced eventually leaving the CD4+ cells weak. Such cells lose the ability to migrate into the tissues from the blood stream via diapedesis, the energetic process whereby cells exit the circulation by pushing themselves between the lining cells of veins and capillaries. Cells too weak to do this must remain in the circulatory system unable to assist calls for assitance from the body tissues.

You can be left with an army of old soldiers who are not able to protect you from the excesses of both the microbes and the younger generation of TH17 cells (the "hairy teenagers") - heavily-armed, aggressive and prone to overdoing things.

Mould toxins can also affect the ability of the CD4+ cells to produce the cytokine proteins necessary to control the TH17 cells. The key cytokines used are Gamma interferon in TH1 cells and IL-4 in TH2 cells. Production of these cytokines can be bloocked by chemicals and heavy metals.

There is another mechanism in auto-immune disease whereby toxic TH1 cells mistakenly call neutrophils into their area by releasing cytokines of the CXC class. Neutrophils are short-lived white blood cells that lack DNA and are loaded with chemical weapons. When they are brought into tissues by mistake they only live for two days before they release their load of chemicals, destroying cells and supporting tissues. Mycotoxins can cause this faulty immune mechanism.

Auto-immune therapies

The writer uses bioresonance technology to treat auto-immune disease. This usually involves bringing any underlying fungal disease under control. It requires a few treatments initially with regular followup necessary to keep the immune system on track.

Avoiding animal meats, gluten, dairy, processed food and drink, chemicals and preservatives can be very beneficial. Anti-inflammatory herbs like tumeric can also be useful.

The great majority of people in western countries have little choice but to try often expensive pharmaceutical drugs for severe disease.

Many patients with auto-immune disease produce too many white blood cells. The drug methotrexate is a metabolic poison that works by reducing the overall production of white blood cells. This runs the risk of producing an over-suppressed immune system when the body is exposed to other toxicity. This can lead to opportunistic infections.

Modern drugs work by restricting the movement of T cells into tissues from the lymph glands where they reside or by blocking the action of inflammatory messaging proteins like TNFalpha.

Important Note: Michael finds that some people do better with a mix of immune-suppressive drug therapies and carefully-targetted bioresonance treatment. It is not a matter of one or the other.